Talk:L-741,626

From Neuroleptic article, Wikipedia All antipsychotic drugs tend to block D2 receptors in the dopamine pathways of the brain. This means that dopamine released in these pathways has less effect. Excess release of dopamine in the mesolimbic pathway has been linked to psychotic experiences. It has also been proven[citation needed] less dopamine released in the prefrontal cortex in the brain, and excess dopamine released from all other pathways, has also been linked to psychotic experiences, caused by abnormal dopaminergic function as a result of patients suffering from schismatic or bipolar disorder. Various neuroleptics such as haloperidol and chlorpromazine suppress dopamine chemicals throughout its pathways, in order for dopamine receptors to function normally. This information leads me to believe that this substance would be the perfect neuroleptic: Acting without side-effects on the primary cause of concern over Psychosis, agonizing only this receptor. If some company can license, test, and label this drug, it could really become a miracle and put a lot of Medicaid patients back to work. No stress, no headaches: all that remains is executive function (IQ.) and personal choices. 173.14.238.114 (talk) 01:35, 24 October 2013 (UTC)