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Arthur M. “Buzz” Brown, M.D., Ph.D. is ChanTest’s founder, Executive Chairman & CSO, and an Adjunct Professor of Physiology and Biophysics at Case Western Reserve University, School of Medicine.

He is a trained cardiologist and physiologist with more than 30 years of experience in ion channels and their relationship to human disease and has published extensively in peer-reviewed journals.

Dr. Brown has established world-leading Physiology department at University of Texas Medical Branch, Baylor College of Medicine and Case Western Reserve University.

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Honors and contributions
Dr. Brown’s honors include the following:
 * Recipient of the prestigious Distinguished Service Award by the Safety Pharmacology Society (SPS) in 2013
 * Established Investigator of the American Heart Association
 * Javits Neuroscience Investigator Award
 * The Harry B van Dyke Awards for Excellence in Medical Research (Pharmacology), Columbia University College of Physicians and Surgeons
 * American Physiological Society Water B. Cannon Lecturer
 * Recipient of American Heart Association Basic Research Prize
 * Recipient of Louis and Artur Lucian Award for Research in Circulatory Diseases
 * First Robert M. Berne Distinguished Lecturer of the American Physiological Society Cardiovascular Section
 * Newton-Abraham Visiting Professor in Medical, Biological and Chemical Sciences, (MA Honors), University of Oxford, England

Major contributions from his research laboratories to the field of ion channels include: Dr. Brown is currently a collaborator with the US Federal Drug Administration (FDA) on cardiac safety issues. He holds nine patents on ion channel pharmacology, therapeutics and devices.
 * Invention of ion-selective liquid exchanger microelectrodes ;
 * Description of membrane–delimited G protein effects on ion channels ;
 * Identification of the pore of voltage-gated and inwardly rectifying potassium channels ;
 * Specification of hERG potassium channel as lethal target for non-cardiac drugs, e.g., Seldane, Propulsid, etc.
 * Discovery of drug-induced block of hERG trafficking as cause of arrhythmias.