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Melitta Schachner Camartin (born 4 April 1943 in Brno) is an emerita professor for neurobiology and guest scientist at the Center for Molecular Neurobiology Hamburg at Hamburg University.

Contents

 * 1 Life
 * 2 Research
 * 3 Awards
 * 4 Selected Publications
 * 5 References

Life
Melitta Schachner studied biochemistry at the University of Tübingen, Germany, from 1963-1968. During her studies she worked as a „summer student“ in the lab of Max Delbrück at the California Institute of Technology, Pasadena, USA, (1965), and in the lab of François Gros  at the Institut de Biologie Physico-Chimique, Paris (1966). She obtained her PhD at the Max Planck Institute for Biochemistry in Munich, Germany, in 1970. From 1970 – 1972 she joined the Faculty for Neuropathology at Harvard Medical School in Boston, USA, as a postdoc and spent a sabbatical for research at the Sloan-Kettering Institute in New York, USA in 1971. She was lecturer in neuropathology at the Faculty for Neuropathology at Harvard Medical School in Boston, USA, from 1973-1974, and Assistant Professor of Neuropathology from 1974-1976. 1976 she became Full Professor of Neurobiology at the University of Heidelberg, Germany. From 1976 – 1988 she was Professor of Nneurobiology at the Swiss Federal Institute of Technology in Zurich, Switzerland. Since 1997 Schachner Camartin is a Professor of Neurobiology at the Centre for Molecular Neurobiology, University of Hamburg, Germany.

Currently, she is heading her emeritus laboratory in Hamburg, leading a research group at Rutgers University as New Jersey Professor of Spinal Cord Research, and consulting at the Center of Neuroscience of the Li Kashing University Medical College in Shantou.

Research
Melitta Schachner‘s research is focussed on the role of adhesion molecules in cell interactions of the nervous system. She introduced the antibody technology to discover and functionally characterize adhesion molecules during ontogenetic development, their functional role  in synaptic function and plasticity as well as during nervous system disease and regeneration. Her work showed that adhesion molecules are important for cell cell or cell-matrix recognition and furthermore  affect cellular signaling cascades of neural cells.

Awards
tbd