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Diagnosis
Diagnosis is similar, but slightly different for each type of PD. As well some types are more understood than others.

Paroxysmal Kinesigenic Dyskinesia (Paroxysmal Kinesigenic Choreoathethosis)
The guidelines for diagnosing Paroxysmal Kinesigenic Dyskinesia (also called Paroxysmal Kinesigenic Choreoathetosis) were reviewed and confirmed by Unterberger and Trinka. PKD consists of unexpected forms of involuntary movements of the body. These movements can include dystonia, chorea, and athetosis, which are all different sudden contractions of the muscles. These involuntary movements can be triggered by quick, sudden, but voluntary movements, such as standing up after sitting for awhile, running quickly, or being startled. Many of these PKD patients experience an aura-like sensation or a tingling in their limbs before any spasms occurs. The limbs, body, face, and neck can all be affected, however, the limbs are the most susceptible. As well the spasms usually only last about 1 minute or less, but longer attacks do occur. One of the most defining features about PD compared to epilepsy is that all the patients never lose consciousness during an attack. The patient is usually diagnosed sometime before their 20's. More likely diagnosed during childhood than early adulthood. Almost all PKD's are idiopathic, as well there is a familial history of autosomal dominant inheritance. Physical examination and brain imaging examinations show normal results, and an EEG shows no specific abnormality as well. However, the negative synchronous EEG results can be used to prove that PKD is not a sort of reflex epilepsy but a different disease.

Paroxysmal Non-kinesigenic Dyskinesia
Paroxysmal Non-kinesigenic Dyskinesia has a set guideline for diagnosis that is slightly different than PKD. PNKD usually occurs unexpectedly, and is not brought on by sudden movements or exercise. Instead the attacks are brought on by stresses such as emotional stress, fatigue, alcohol, or caffeine consumption. The attacks are comprised of dystonia, chorea, and athetosis just like PKD. As well these attacks are mostly of the limbs, and are usually unilateral or asymmetric. What sets PNKD apart from PKD is that the attacks can last anywhere from 4 minutes to 4 hours, but shorter and longer attacks have been reported as well. Just like PKD, PNKD also shows autosomal dominance in family history. Physical examination and brain imaging examinations show normal results, and EEG shows no specific abnormality as well.

Paroxysmal Exercise-induced Dyskinesia
Paroxysmal Exercise-induced Dyskinesia had a set guideline for diagnosis that is similar, but slightly different than both PKD and PNKD. PED attacks are comprised of dystonic and bilateral movements usually in the lower limbs of the body. These attacks are brought about only by exercise and physical exhaustion. PED patients do not feel an aura-like sensation before an attack occurs, unlike PKD and PNKD. As well, these attacks usually last from 5 to 30 minutes, and can occur once a day or once a month. Physical examination and brain imaging examinations show normal results, and EEG shows no specific abnormality as well.

Prognosis
Paroxysmal Dyskinesia is not a fatal disease. Life can be extremely difficult with this disease depending on the severity. The prognosis of PD is extremely difficult to determine because the disease varies from person to person. The attacks for PKC can be reduced and managed with proper anticonvulsants, but there is no particular end in sight for any of the PD diseases. PKC has been described to cease for some patients after the age of 20, and two patients have reported to have a family history of the disease where PKC went into complete remission after the age of 23. With PNKD and PED, at this time, there is no proper way to determine an accurate prognosis.