User:Gcarpen7/Choose an Article

Article Selection
Please list articles that you're considering for your Wikipedia assignment below. Begin to critique these articles and find relevant sources.

Option 1

 * Article title
 * CDw17 antigen


 * Article Evaluation
 * There is not much here, but I am finding plenty of resources that can be used to elaborate on this topic.
 * Sources
 * -Involvement of glycosphingolipid-enriched lipid rafts in inflammatory responses
 * -ST3GAL5 - Lactosylceramide alpha-2,3-sialyltransferase - Homo sapiens (Human) - ST3GAL5 gene & protein (uniprot.org)
 * -B3GNT5 - Lactosylceramide 1,3-N-acetyl-beta-D-glucosaminyltransferase - Homo sapiens (Human) - B3GNT5 gene & protein (uniprot.org)

Option 2

 * Article title
 * Emopamil
 * Article Evaluation
 * This calcium channel blocker only has one sentence for its page, yet many articles come up with a basic search.


 * Sources
 * Emopamil - an overview | ScienceDirect Topics

Option 3

 * Article title
 * Kallidin


 * Article Evaluation
 * This article states the bioactive kinin is created upon injury, but is vague. The chemicals anti-inflammatory responses may be elaborated upon.


 * Sources
 * Evidence for activation of the tissue kallikrein-kinin system in nociceptive transmission and inflammatory responses of mice using a specific enzyme inhibitor

Option 4

 * Article title
 * RAI1


 * Article Evaluation
 * Gives a brief about RAI1 being transcription factor in relation to a few syndromes.


 * Sources
 * RAI1 gene: MedlinePlus Genetics
 * RAI1 Gene - GeneCards | RAI1 Protein | RAI1 Antibody

Option 5

 * Article title
 * Killin KLLN (gene)


 * Article Evaluation
 * There is no article for this one but perhaps there should be. This gene, when mutated, causes Cowden's syndrome and is aides in apoptosis.  The gene is being researched for cancer fighting properties.


 * Sources
 * KLLN Gene - GeneCards | KILIN Protein | KILIN Antibody
 * Androgen receptor-induced tumor suppressor, KLLN, inhibits breast cancer growth and transcriptionally activates p53/p73-mediated apoptosis in breast carcinomas