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Mind blindness is a condition where a person in unable to attribute to mental states. Mind blindness is usually associated with people that have been diagnosed with Autism and Asperger's syndrome.

Improvements and additions for the article
Elaborate on the introduction(give some more information on its prevalence and briefly mention it effects. Origins of mind blindness. (Frith 2001, Galagher and frith 2003, S baron-cohen 2012 and 1990)

Give some more background information of the theory of mind (what is it, how and when it develops in children). Extend this to mind blindness. ( Lombardo and Cohen 2010, frith 2001, Shanker 2004, Bowler 1992, Colle Cohen and Hill 2007, Perner and Lang 1999)

Biological basis of the theory of mind and mind blindness. How it develops, mechanisms involved (Gallagher and Frith 2003, Thomassin et al 2006, Cohen 2012, bird et al 2004-Looking for more sources)

Give more indicators of mind blindness and how it is associated with asperger’s syndrome and autism. (Cohen et al 2005, Cohen 1990, Cohen 1989, Bowler 1992, Colle Cohen and Hill 2007, Dorris et al 2004, Klin et al 1992)

Emphasize more on the E-S theory and how it is generally accepted amongst researchers. Cognitive aspects of mind blindness in patients. Mention this in the criticisms (Simon Baron-Cohen, Wheelwright, Lawson, Griffin, and Hill, Cohen et al 2005)

Elaborate on the criticisms. How it is not unique, flaws and loopholes in the theory (Shanker 2004-Looking for more sources)

Introduction
Mind Blindness-general information (symptoms, effect, disorders associated with it, Simon-Baron Cohen)

Mind Blindness in relation to Theory of Mind

Details-Relevance, causes
Biological Basis of Mind Blindness

Mind Blindness in relation to Autism/Asperger’s/Schizophrenia

Causes-Mechanisms involved

E-S Theory-Alternative to current beliefs

Conclusion
Criticisms

Article
Wikipedia Assignment Mind-Blindness can be described as a cognitive disorder where an individual is unable to attribute mental states to the self and other. As a result of this disorder the individual is unable to mind read. The individual is also not capable to have any sort of beliefs and desires (frith and galgher 2003). The beliefs and desires stem from expectations of an individual, those with mind-blindness are unable to generate expectations and predict behaviours. This ability to develop a mental awareness of what is in the mind of an individual is knows as the theory of mind. This allows one to attribute our behaviour and actions to various mental states such emotions and intentions. Mind-blindness is associated with autistic and asperger’s syndrome patients. Patients tend to show deficits in social insight. The mind-blindness hypothesis was termed to explain these deficits in autistic patients (frith 2001

In the 1960s scientists were unable to agree on what austim or asperger’s syndrome was. It was debated upon then whether autism and asperger’s patients suffered from developmental language disorders or emotional disorders. This paved way for the mind-blind hypothesis that was based on theory of mind. ) ---Include latter part of para 1

Mindblindness and Theory of Mind Mind-blindness is a state where the theory of mind has not been developed or lost in an individual. The theory of mind is implicit in the normal individuals. This enables one to make automatic interpretations of events taking into consideration the mental states of people, their desires and beliefs (frith2001). An alternative approach to the social impairment observed in mindblindness focuses on emotion of subjects. Based on empirical evidence, Frith concluded that the processing of complex cognitive emotions is impaired compared to simpler emotions. According to Frith, attachment does not seem to fail in the early childhood of autistics. It is beleived that emotion is a component of social cognition that is separable from metalizing.

Biological Basis of Mind-blindness Neural correlates of the theory of mind point towards three regions of the brains, the anterior paracingulate cortex (Broadmann),The anterior paracingulate cortex is considered at the key region of mentalizing. It is located anterior corpus callosum and the anterior cingulated cortex. This cortex is associated with the medial frontal cortex where activation is associated with the mentalization of states. However it is still debated whether the inactivation of the medial frontal lobe is involved in the mind-blindness.

In addition to the anterior paracingulate cortex is the superior temporal sulcus and the temporal poles. These areas are not uniquely associated with mentalization. They aid in the activation of the regions that are associated with the theory of mind. The superior temporal sulcus is involved in the processing of behavioural information while the temporal poles are involved in the retrieval of personal experiences. These are considered important regions for the activation of the theory of mind regions and are associated with the mind-blindness. The temporal poles provide personal experiences for mentalization such as facial recognition, emotional memory and familiar voices. In patients suffering from semantic dementia, the temporal regions of these patients undergo atrophy and lead to certain deficits which can cause mind-blindness.

The amygdala and the orbitofrontal cortex also are a part of the theory of mind. It is in involved in the interpretation in of behaviour which plays an important role in social cognition and therefore contributes to the theory of the mind. Damage to the orbitofrontal cortex brings upon subtle impairments but not a total loss in the theory of mind leading to mind-blindness. Numerous studies over the years have shown that the orbitofrontal cortex is not directly associated with the theory of the mind or mind-blindness. Lesion studies, show that when lesions are given to the medial frontal lobe, performance on mentalization tasks is reduced, similar to mind-blindness. Patients that experienced frontal lobe injuries due to severe head trauma showed signs of mind blindness, as a result of a lost theory of mind.

Autism and Asperger’s and mindblindness Mind-blindness is associated is usually seen with patients suffering from pervasive developmental disorders such as autism and asperger’s syndrome. Cohen proposed the mindblindness theory of autism as “deficits in the normal process of empathising”. He described empathising to include the theory of mind, mind reading and taking an intentional stance. According to this view empathising includes the ability to attribute mental states and to react in an appropriate emotional manner which is appropriate to another’s mental state. According to Baron-Cohen in 1989, more deficits tend to occur in reference to one’s own mental states compared to the other’s mental states. It has been proposed that autistic people undergo a specific developmental delay in the area of metarepresentational development. The delay facilitates mindblindness. (braon cohen 2012) However it is speculated that the theory of mind develops occurs in a continuum. There is some evidence that suggests that certain patients develop a rudimentary theory of mind and do not suffer from complete lack of theory of mind causing mind-blindness.

The social and cognitive impairments seen in patients suffering from pervasive developmental disorders can be attributed due to mind blindness. Abnormal behaviour of children in autism includes the lack of reciprocity. Sever cases in which mind-blindness manifests includes the child being totally withdrawn from social settings while in less severe cases the individual may attempt to interact with other people. Individuals suffering from more severe forms of autism tend to not make eye contact and are unable to engage in simple social tasks. Despite this sever form of autism that is characterised by social detachment, global asocial behaviours is not the rule in autism. Cohen described the cognitive/mindblindness effects in autistic patients as a “triad of deficits”. The triad consists of deficits in social, communication and imagination of others’ mind (cohen 2012).

Ozonoff and colleagues were able to discriminate between individuals suffering from Asperger’s syndrome and autistics by their ability to solve theory of mind tasks. It is due to patients suffering from AS undergo fewer abnormalities in development early on in childhood. The siblings of individuals diagnosed with Tom were shown to have a lesser variant of theory of mind deficits. This shows that the cognitive deficits that affect the theory of mind play central role in the phenotype expressed in AS patients.