User:Hunterw97/sandbox

Hello Hunter,

I think you have a good start on your addition to your page. I would suggest reading it over again to check for grammar and punctuation errors. If you would like, I can go through and highlight some areas. In one sentence you seemed to use the word "gene" a lot. Is there any way to mix that sentence up so that word is not so frequent? You seemed to not touch on some of your content gaps listed. Did you branch off to a different addition, or will there be any other changes to mend this?

Thank you,

Jaden

Thank you Jaden for the review I will expand my work and check more in depth for grammar mistakes.

Hi Hunter it's Shannon, hope this video helps!

Resources for wiki A simple and cost-effective method for screening of CRISPR/Cas9-induced homozygous/biallelic mutants Authors: http://eds.b.ebscohost.com/eds/detail/detail?vid=0&sid=82366751-e4e2-4629-ab79-16c802d63e25%40pdc-v-sessmgr02&bdata=JkF1dGhUeXBlPXNoaWImc2l0ZT1lZHMtbGl2ZQ%3d%3d#AN=edsdoj.4394ad190b704133b92957c596301f66&db=edsdoj

Evaluation of Drought response in Some Rice Mutant Lines Using Stress Tolerance Indices http://eds.b.ebscohost.com/eds/detail/detail?vid=0&sid=b395387a-3fb1-4d67-9923-984819545965%40pdc-v-sessmgr02&bdata=JkF1dGhUeXBlPXNoaWImc2l0ZT1lZHMtbGl2ZQ%3d%3d#AN=edsdoj.8ddb49d2dc19412a8515cf88f537dd35&db=edsdoj

The truncated mutant HBsAg expression increases the tumorigenesis of hepatitis B virus by regulating TGF-β/Smad signaling pathway http://eds.b.ebscohost.com/eds/detail/detail?vid=0&sid=df9b906a-516e-4015-bcf2-3db887c16244%40pdc-v-sessmgr05&bdata=JkF1dGhUeXBlPXNoaWImc2l0ZT1lZHMtbGl2ZQ%3d%3d#AN=edsdoj.b4bf885f03904db9bfa660a2549602e8&db=edsdoj

Content gaps in Mutant. 1. An explanation that goes with a pictorial image of a mutated virus RNA to show how the next virus although very similar is not the same as the original but is now a mutant. 2. an idea of how common mutants are in some species that are externally visible like the blue lobster. 3. if possible a pictorial image of dna repair missing a damaged area which moves on to produce the mutant. 4. if a mutation is missed by DNA repair how does it affect the organism as a whole. 5. example of organism that has undergone mutation and is as a mutant better suited for it's environment. 6. When reproducing how well does mutant's dna interact with another mutant's dna. aka will another's be able to silence a mutant's gene or will they simply merge to make yet another mutant keeping both's mutation. 7. are there notable studies with artificial mutants? 8. identifying some non carcinogenic mutagens. 9. there is no referance of a mutant coming about due to radiation that is not cancer.

content gap: explain how mutant in and experiment is not a mutant even if it is mutating due to something found in it's natural environment. it is not a true mutant because it did not occur in the wild

to go at the end of mutants by mutation. Chimeric DNA/RNA can be used on mammalian cells to make chromosomal changes in which can cause a mutant. Some plant cells can also be modified by the same method such that the chimeric DNA/RNA can cause drastic changes like herbicide resistance. As long as DNA repair is not able to correct the mutation the cell will go on as a mutant cell. In the inverse effect A chimeric gene can correct a gene because of the ability to target that gene. E, Coli was introduced to a chimeric plasmid that caused antibiotic resistance. Through testing tomato plants under conditions of increased salinity through irrigation key genes were discovered which allow the plants to tolerate the conditions.