User:Samuel Raaflaub/Klüver–Bucy syndrome

Article Draft
Klüver–Bucy syndrome is more commonly found in rhesus monkeys, where the condition was first documented, than in humans. Pathology on the syndrome is still controversial with Norman Geschwind's theory and Muller theory offering different explanations for the condition. Treatment for Klüver–Bucy syndrome is minimal with no current cure.

Pathology
There are a few theories that attempt to explain the processes behind Klüver–Bucy syndrome and its symptoms. This topic still remains controversial as complete Klüver–Bucy syndrome is rare in humans especially compared to monkeys. Klüver–Bucy syndrome is thought to occur from damage to temporal sections of the limbic networks, which connects to other structures that regulate emotional behavior. Norman Geschwind's theory states that Klüver–Bucy syndrome is caused by disconnect syndrome (a condition of the brain where the two hemispheres develop separately or at different rates) and that the initial contributor for this is the blockage of visual input to the limbic circuit. Another theory, called Muller theory, attributes Klüver–Bucy syndrome to the disconnection of pathways used for emotional regulation and memory, such as those connecting the dorsomedial thalamus to the prefrontal cortex. The medial temporal sections of the limbic system can be associated with more primitive functions such as reproduction, food, and defence. This can be seen in the symptoms of increased hypersexuality, hyperorality, and general aggression.

Treatment/Management
Treatment for Klüver–Bucy syndrome focuses on controlling the present symptoms because no current intervention will cure the condition. Medications used to treat the symptoms include carbamazepines and leuprolide, which help reduce sexual behavior associated with Klüver–Bucy syndrome. For carbamazepines it has been shown to have the greatest effect when treating traumatic brain injury-derived Klüver–Bucy syndrome. Other medications commonly utilized are mood stabilizers, antidepressants, and various antipsychotic drugs.

In Children
Klüver–Bucy syndrome has been shown to occur more in adults than in children. In children certain conditions such as herpes simplex encephalitis has the potential to manifest the syndrome. Children exhibit many of the same symptoms as adults with Klüver–Bucy syndrome, however they display these symptoms in different ways than adults. Hypersexualtity, as a symptom of Klüver–Bucy syndrome, in children is characterized by "frequent touching of genitals, intermittent pelvic thrusting movements, and rubbing of genitals on the bed while lying prone". Observations of hypersexuality in children were recorded from children primarily under the age of 4. It is thought that since these behaviors have yet to be learned by children that they display themselves in more ambiguous ways. The underlying conditions that lead to Klüver–Bucy syndrome can have an effect on the pattern of symptoms observed.

I am not sure why you have so many numbers in advance of the citation (e.g., 1.0, 1.1, 1.2)?

''Suggested edits throughout in italics and parentheses. (Liz)''

Summary of Peer Review
For the peer review I looked over the grammar and sentence structure of my article and opted to change inaccuracies. I was linked an article however I am still debating on where or not to use the article because it is mostly about hyperorality ( a specific symptom) rather that the syndrome.