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WikiProject Medicine/Translation task force/RTT/Simple Asperger syndrome (March 13, 2016)

Asperger syndrome (AS) is a developmental disorder characterized by significant difficulties in social interaction and nonverbal communication, along with restricted and repetitive patterns of behavior and interests. It is an autism spectrum disorder (ASD) and differs from other disorders by relatively normal language and intelligence. Although not required for diagnosis, physical clumsiness and unusual use of language are common. Symptoms usually begin before two years old and can last for a person's entire life.

The exact cause of Asperger's is unknown. While there is likely a genetic basis it has not been determined. Environmental factors are also believed to play a role. Brain imaging have not identified a common underlying problem. The diagnosis of Asperger's was gotten rid of in the 2013 fifth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-5) and people with these symptoms are now included within the autism spectrum disorder along with autism and pervasive developmental disorder not otherwise specified. It remains within the tenth edition of the International Classification of Diseases (ICD-10) as of 2015.

There is no single treatment, and the effectiveness of particular interventions is supported by only limited data. Treatment is aimed at improving poor communication skills, obsessive or repetitive routines, and physical clumsiness. Efforts may include social skills training, cognitive behavioral therapy, physical therapy, speech therapy, parenting training, and medications for associated problems such as depression or anxiety. Most children improve as they grow up, but social and communication difficulties may persist. Some researchers and people on the autism spectrum have advocated a shift in attitudes toward the view that autism spectrum disorder is a difference, rather than a disease that must be treated or cured.

In 2013, Asperger's was estimated to affect 31 million people globally. The syndrome is named after the Austrian pediatrician Hans Asperger who, in 1944, described children in his practice who lacked nonverbal communication, had limited understanding of others feelings, and were physically clumsy. The modern conception of Asperger syndrome came into existence in 1981 and went through a period of popularization. It became a standardized diagnosis in the early 1990s. Many questions and controversies remain about aspects of the disorder. There is doubt about whether it is distinct from high-functioning autism (HFA). Partly because of this, the percentage of people affected is not firmly established.

WikiProject Medicine/Translation task force/RTT/Simple Dementia with Lewy bodies (October 3, 2016)

Dementia with Lewy bodies (DLB) is a type of dementia that gradually worsens over time. Additional symptoms may include fluctuations in alertness, seeing things that other people do not, slowness of movement, trouble walking, and rigidity. Excessive movement during sleep and mood changes such as depression are also common.

The cause is unknown. There is typically no family history among those affected. The underlying mechanism involves the buildup of Lewy bodies, clumps of alpha-synuclein protein in neurons. It is classified as a neurodegenerative disorder. A diagnosis may be suspected based on symptom, with blood tests and medical imaging done to rule out other possible causes. The differential diagnosis includes Parkinson's and Alzheimer's.

There is no cure for DLB. Treatments try to improve mental, psychiatric, and motor symptoms. Acetylcholinesterase inhibitors, such as donepezil, may provide some benefit. Some motor problems may improve with levodopa. Antipsychotics, even for hallucination, should generally be avoided due to side effects.

DLB is the most common cause of dementia after Alzheimer's and vascular dementia. It typically begins after the age of 50. About 0.1% of those over 65 are affected. Males appear to be more commonly affected than females. In the late part of the disease people may depend entirely on others for their care. Life expectancy following diagnosis is around 8 years. The abnormal deposits that cause the disease were discovered in 1912 by Frederic Lewy.

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