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Involvement of capsaicin-sensitive nerves in regulating the hormone and glucose metabolic response to endotoxin: The study indicates that capsaicin intakes destroy primary sensory afferent nerve fibers, which cause LPS-induced increase in a significant number of species, and later appeared hypoglycemic.

Effect of capsaicin-sensitive sensory nerves on plasma glucose and catecholamine levels during 2-deoxyglucose-induced stress in conscious rats.: When capsaicin was injected to the mouse, hypoglycemic reaction occurred in respond to 2-deoxyglucose (2-DG), which indicates that capsaicin-sensitive sensory fibers regulate plasma glucose level when glucopenic stress is present.

Capsaicin sensitive afferent neurons from peripheral glucose receptors mediate the insulin-induced increase in adrenaline secretion.: Capsaicin allow insulin to drop the blood glucose concentration, which reduces the urinary adrenaline excretion for over 24 hours.

Capsaicin-sensitive sensory neurons are involved in the plasma catecholamine response of rats to selective stressors.: Capsaicin-sensitive sensory neurons induce plasma CA response, which responds to cold stress.

Reflist:
Amann, R. & Lembeck, F. Naunyn-Schmiedeberg's Arch. Pharmacol. (1986) 334: 71. https://doi.org/10.1007/BF00498742 Morgan, A.E., Lang, C.H. Involvement of capsaicin-sensitive nerves in regulating the hormone and glucose metabolic response to endotoxin. ''Am. J. Physiol.'' 1997;273:E328–E335. Zhou, X F, and B G Livett. “Capsaicin-Sensitive Sensory Neurons Are Involved in the Plasma Catecholamine Response of Rats to Selective Stressors.” The Journal of Physiology 433 (1991): 393–407. Print. Zhou, X. F., and B. G. Livett. “Effect of Capsaicin-Sensitive Sensory Nerves on Plasma Glucose and Catecholamine Levels during 2-Deoxyglucose-Induced Stress in Conscious Rats.” British Journal of Pharmacology 100.3 (1990): 523–529. Print.