User talk:Drimmuno

Everyone has been inquiring about how the COVID-19 virus kills. While there are many symptoms of this virus, the CDC continues to add new symptoms to indicators of the COVID-19 virus. There have been numerous reports that the COVID-19 virus is more lethal in elderly patients. This is because people, in general, produce higher amount of ACE-2, the ligand (cell-adhesive molecule), the COVID-19 virus attaches to. Since the younger population has less ACE-2 than their elderly peers, they are less likely to show symptoms. Unfortunately, because elderly patients produce more ACE-2, they are more vulnerable. The weird thing about viruses, with the COVID-19 virus being indifferent, are there random symptoms that they bring on. While random may not be the appropriate word, the main point is the list of symptoms that the CDC advertises as symptoms of the COVID-19 virus may vary from person to person, with different patients having different symptoms showing first (e.g., a headache, fever, sore throat, cough, etc.)

While few youth patients may experience similar symptoms to their elderly peers, they can still experience deadly symptoms. Albeit the odds of death in youth patients is dramatically inferior to the odds of death for an elderly patient, it, unfortunately, still happens. Several factors to why pediatric and youth patients may die pertain to their overall health, ACE-2 levels, and how good of "condition" (whether they are in homeostasis) when they catch the virus or not. In rare instances, perfectly healthy individuals may die of COVID-19 due to the fact that they have a mutations such as the RAB1A, KDM5B, HAT1, and HDAC2.

Unfortunately, it is well known that the sicker the patient is, the more likely they are to die from the illness causing their troubles. The COVID-19 virus is indifferent. Ligands on the virus, also known as glycoproteins, also latch onto hemoglobin. As patients start to lose oxygenation, or their O2 stats dwindle, they are at a higher risk of death. The reason for this is because the body relies on oxygen to not only live, but to carry out essential functions to keep us alive. One of these critical requirements to stay alive is the ability for the body to deliver a "powerful punch" to any foreign invaders (i.e., COVID-19 virus). Additionally, COVID-19 bonds to a protein called hemoglobin, a protein responsible for carrying oxygen in the blood of many animals, including humans. As patients lose oxygen, the hemoglobin goes from an oxygenated state to a deoxygenated state. When hemoglobin is deoxygenated, it is especially vulnerable to COVID-19 attack. The oxygen is replaced with the virus, furthering its spread and increasing the likelihood of mortality. When COVID-19 attaches to deoxygenated hemoglobin, the hemoglobin molecule is disabled and less capable of delivering oxygen to vital areas of the body. This catalyzes, or speeds up death. When cells lose vital oxygen, the cells start to die. When the cells die, less and less cells are able to accomplish the function established by the body's DNA. As more and more cells start to die, the brain inevitably recognizes it receives less and less nutrition and oxygen. Hence, the brain realizes it should shut down. This is where patients are put on ventilators. Ventilators, more often than not, are the last stage/efforts for physicians to fight the virus and keep the body alive. This is done, optimistically, by giving letting the brain receive more oxygen to direct the body to mobilize a stronger response. While some patients will improve after a few weeks on ventilator, many die because the brain is further disabled by the virus' efforts to thwart the body's effective hemoglobin levels. Once the brain has a certain amount of cells die, it, more often than not, irrevocably kills itself. The result is the patient's heart, lungs, and body shuts down.