User talk:Immcarle74/sandbox

Bibliography of sources:

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Romee, R., Foley, B., Lenvik, T., Wang, Y., Zhang, B., Ankarlo, D., Luo, X., Cooley, S., Verneris, M., and Walcheck, B. et al. (2013). NK cell CD16 surface expression and function is regulated by a disintegrin and metalloprotease-17 (ADAM17). Blood 121, 3599-3608.

Sánchez-Torres, C., García-Romo, G., Cornejo-Cortés, M., Rivas-Carvalho, A., and Sánchez-Schmitz, G. (2001). CD16+ and CD16− human blood monocyte subsets differentiate in vitro to dendritic cells with different abilities to stimulate CD4+ T cells. International Immunology 13, 1571-1581.

Wu, J., Edberg, J., Redecha, P., Bansal, V., Guyre, P., Coleman, K., Salmon, J., and Kimberly, R. (1997). A novel polymorphism of FcgammaRIIIa (CD16) alters receptor function and predisposes to autoimmune disease. Journal Of Clinical Investigation 100, 1059-1070.

Yeap, W., Wong, K., Shimasaki, N., Teo, E., Quek, J., Yong, H., Diong, C., Bertoletti, A., Linn, Y., and Wong, S. (2016). CD16 is indispensable for antibody-dependent cellular cytotoxicity by human monocytes. Scientific Reports 6.

Immcarle74 (talk) 20:19, 29 January 2018 (UTC)