Wikipedia:Peer review/Attention deficit hyperactivity disorder/archive1

Attention deficit hyperactivity disorder
This peer review discussion has been closed. I've listed this article for peer review because I plan to work on this for FA once the amphetamine FAC closes and I finish editing this article and GA nominate it (I'll probably work on ADD for GA as well). I'd just like some input from others on potential improvements to style, citations, and scope/coverage. I plan to use the feedback from this review as, more or less, a to-do list for when I'm ready to work on the article.

Thanks,  Seppi  333  (Insert 2¢) 21:43, 17 January 2014 (UTC)


 * I just took a very quick glance, and think it is going to take a sustained effort to bring this article close to FA standards. Sometime since the last time I looked, some poorly sourced text calling Tourette syndrome a "rare" (not) disorder crept in, and tics were called "nervous" tics.  The relationship between ADHD and TS is the subject of scores and scores of journal reviews, and is basically not covered here, and the contentious relationship isn't adequately covered in the faulty info from the NIH factsheet. There is scant coverage of the serious amount of information known about the neuropsych profile associated with ADHD, and the Treatment section is cursory at best.  Prognosis also needs a lot more beef.  To work this article up, someone needs to get hold of several dozen good reviews, and the article will need to depend on summary style, with multiple sub-articles.  "Rare" disorder, "nervous" tics ... the NIH has long had bad information on their factsheet pages, and this has been discussed several times at WT:MED-- ditch them-- there are so many high-quality secondary reviews that sources like that shouldn't be used.  Sandy Georgia  (Talk) 14:29, 18 January 2014 (UTC)
 * I expect to have my work cut out for me, but have a ton of relevant reviews and meta-analyses on ADHD or stimulants for ADHD that I plan to use from work on other articles.  Seppi  333  (Insert 2¢) 21:04, 18 January 2014 (UTC)

Notes (to self) by Seppi333 on potential sources for inclusion
Please add new comments/sections above this section.

Note to self:convert month and year to date This section is mostly just for my convenience for reference and as a pre-formatted source list. I plan to use some/all of these later (this list will expand):


 * Relevant general material on ADHD or ADHD stimulants from Amphetamine


 * Material on ADHD from Molecular Neuropharm

Outlined references:
 * ADHD treatment in specific groups:
 * Athletes:
 * Review
 * Review
 * Children & Young adults:
 * Review
 * Review
 * Comorbid SUD:
 * Review
 * Review
 * Stims
 * Safety:
 * Review
 * Efficacy:
 * Review
 * Comparison:
 * Meta-analytic systematic review
 * Supplemental phospholipids: (I expect to write at most a paragraph on this)
 * Phosphatidylserine w/ DHA/EPA - effect on protein kinase C signaling and transporter phosphorylation
 * Review
 * Potential review? (check article ADHD coverage for applicability)
 * RCT's on this topic published after the most recent review:
 * RCT+extension
 * RCT
 * RCT w/ PS alone
 * Insufficiency of DHA/EPA alone:
 * Review
 * Trace amine neuromodulation and ADHD
 * 3 Reviews

Copied from Talk:Amphetamine/Archive 4
Every primary source in this paragraph could be replaced with one of the following 3 reviews:

In individuals with ADHD, there is significant evidence that phenethylamine (PEA) – an endogenous amphetamine homologue with analogous dopaminergic pharmacodynamics – metabolism is reduced compared to healthy individuals. It is well documented that urinary excretion of PEA increases following administration of amphetamine and methylphenidate, and that urinary excretion of these drugs is highly correlated with urinary excretion of PEA;  moreover, studies on rodents show that brain PEA biosynthesis and metabolism greatly increases following amphetamine administration at therapeutic doses. There is also evidence that pharmacological depletion of PEA blocks the stimulant effects of amphetamine, suggesting that endogenous PEA plays an important role in mediating the effects of amphetamines. (Note to self: Update the text of the struck-out clauses to current evidence on humans from these reviews)

Uncategorized:

— Seppi  333  (Insert 2¢)