Talk:Stephen Faraone

Section of primary references
This section was based off of citations to primary sources. My logic for removing it is here. Research Faraone’s studies of the genetic epidemiology of attention-deficit hyperactivity disorder (ADHD) focused on the establishing patterns of familial transmission in families ascertained through ADHD boys and girls with colleagues at the Massachusetts General Hospital and Harvard Medical School. His subsequent studies clarified the genetic heterogeneity of that disorder. He has shown that 1) ADHD with conduct or bipolar disorders is a genetically homogeneous subtype of ADHD;      and 2) major depression is a nonspecific expression of ADHD genotypes. He has also shown that the form of ADHD that persists into adolescence is highly heritable, making a suitable candidate for linkage analyses. He used genetic paradigms to provide evidence for the validity of diagnosing ADHD in adults and has used adult samples to confirm the putative association between ADHD and the DRD4 receptor gene and completed a meta-analysis to confirm the validity of the association across many studies.

Faraone obtained funding from the National Institute of Mental Health to chair yearly international meetings of researchers studying the molecular genetics of ADHD. The main goal of these meetings is to foster collaboration (and reduce unproductive competition) among researchers studying the genetics of ADHD.

One line of his research has focused on assessing the validity of the diagnosis of ADHD in adults, which has been a source of much controversy in the scientific and clinical literature about ADHD. His review of the literature supported idea that adult ADHD is a valid disorder and his meta-analysis of ADHD follow-up studies showed that about two-thirds of ADHD youth continue to have impairing symptoms of ADHD in adulthood. He has also addressed psychometric issues from several perspectives. In one paper he presented theoretical issues that suggest ADHD is not a developmentally sensitive diagnosis. In another he showed that having an ADHD child does not bias adults with ADHD to over-report ADHD symptoms. He has also shown that, from a genetic perspective, symptom reporting in adults with ADHD may be more valid than symptom reporting in children. He has also shown using patterns of inheritance, neuropsychological studies, studies of substance use disorders, and studies of personality traits, that the DSM-IV age at onset criterion is too strict and that ADHD may validly onset in adolescence.

Additional published work includes animal model studies of ADHD, family studies of ADHD,           candidate gene studies of ADHD,       meta-analyses of candidate gene studies,   linkage analyses of ADHD,   a genome-wide association study (GWAS) of ADHD  and a pharmacogenetic GWAS of methylphenidate response in ADHD. He has also published methodological papers,   and systematic reviews of the genetics of ADHD,   the neurobiology of ADHD  and environmental risk factors for ADHD.

Perhaps this content will be useful to someone looking for secondary sources about these things. Jesanj (talk) 18:29, 30 October 2011 (UTC)

Job
"Stephen V. Faraone is professor of psychiatry, neuroscience and physiology and director of Research for Upstate’s Department of Psychiatry and Behavioral Sciences. He also holds appointments as senior scientific advisor to the Research Program Pediatric Psychopharmacology at the Massachusetts General Hospital and as lecturer at Harvard Medical School."

Stephen V. Faraone, PhD, named SUNY Distinguished Professor | UpstateOnline: A Publication for Upstate Medical University

--24.97.201.230 (talk) 04:53, 13 April 2014 (UTC)

External links modified
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